Rebuttal To Creationists - "Since We Can't Directly Observe Evolution..."

Kleinman:

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Taq will now tell us all about infectious diseases. He knows all about how bacteria evolve resistance, it's ERVs.

Taq:

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Bacteriophage do contribute to antibiotic resistance.
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Bacteriophages Contribute to the Spread of Antibiotic Resistance Genes among Foodborne Pathogens of the Enterobacteriaceae Family – A Review
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Of course, phage aren't ERV's, but integrated virus nonetheless.

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See, Taq knows all about how bacteria get antimicrobial resistance, he's a genius. Of course, he never explains how the phage gets the resistance allele.

Kleinman:

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It certainly doesn't apply to your magical world of ERVs.

Taq:

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Please show me how my math is wrong for computing the chances of independent retroviral insertions creating orthologous insertions at 99+% of 200,000 ERV's.

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You can believe that 8% of the human genome is ERVs, I don't. That's garbage just like the biologist claim that most of the DNA is junk DNA.

Kleinman:

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You make up populations, you imagine alleles increasing infrequencies, none of your ideas is based on real examples.

Taq:

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What the hell are you talking about? Populations exist. Alleles increase in frequency. Are you really saying none of these things exist in nature?
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I just showed you that your addition rule allows two variants at different loci to increase in frequency together. This is all you have in return?

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You make up populations, you make up advantageous alleles, and you don't do real examples. And when I do real examples, you say they don't apply. You let your imagination run amok.

Kleinman:

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What are you talking about? You can have competition between different species let alone competition in a species. The only requirement is that the replicator uses the same resources.

Taq:

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I am talking about competition within a species. For sexually reproducing species, variants at different loci do not compete with one another.

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In the real world, competition exists between species, not just within species. What do you think insects are doing when they eat a crop grown for humans? You really don't understand competition at all.

Kleinman:

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That's a stupid thing to say. I wrote the mathematics of recombination, dummy.

Taq:

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And yet you still can't figure out how alleles at different loci can increase in lockstep with one another, and why there is no clonal interference in sexually reproducing populations.

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This is your imagination speaking. Why don't you give us a real example? What alleles are increasing in frequencies in lockstep with humans?

Kleinman:

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See, Taq knows all about how bacteria get antimicrobial resistance, he's a genius. Of course, he never explains how the phage gets the resistance allele.

Taq:

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Why should I give you an explanation when all you have is mockery in return?

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Try giving a real example rather than one you make up.

Kleinman:

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You can believe that 8% of the human genome is ERVs, I don't.

Taq:

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I accept the findings in the human genome paper. Why don't you? What is wrong with their methods for counting ERV's?

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The idea of hundreds of thousands of viruses infecting cells and yet not causing disease is nonsense, especially in the initial stem cell of a replicator. You can believe it if you want but I think it is hogwash. This is the kind of garbage suitable for Tany while he is chasing bugs.

Taq:

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There are real world examples of existing retroviruses creating new ERV's in modern populations. The Koala genome is currently undergoing an invasion of retroviral sequence into their genome.

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Sure, HIV is an example. Now imagine someone infected with 100,000 different HIV viruses.

Kleinman:

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The idea of hundreds of thousands of viruses infecting cells and yet not causing disease is nonsense, especially in the initial stem cell of a replicator.

Taq:

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Mutations can occur in the virus just before or during insertion that results in a non-replicating virus. You are assuming that all viral insertions are capable of replication and producing viable virus, which just isn't true.

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You imagine hundreds of thousands of inactivated viruses. None of these viruses are active. They all just happen to get mutations that cause them all to become inactive.

Kleinman:

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You can believe it if you want but I think it is hogwash.

Taq:

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I will take the well informed opinion of the world's virologists and geneticists over your uneducated opinions.

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Sure, they have wiped out viral diseases all over the world. They are so smart. They have done a wonderful job with Covid.

Kleinman:

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Now imagine someone infected with 100,000 different HIV viruses.

Taq:

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Imagine one cell with an insertion incapable of producing virus.
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We have examples of an existing virus that is circulating in koalas. We also have examples of ERV's from that virus in the genomes of koalas. What more do you need

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Just 99,999 to go and they are all inactivated.

Kleinman:

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You imagine hundreds of thousands of inactivated viruses.

Taq:

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No need to imagine them. We can observe them in the human genome and the genomes of many other mammals.

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They don't have to be inactivated if they aren't viruses. But you believe they are. You have quite an imagination.

Kleinman:

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Just 99,999 to go and they are all inactivated.

Taq:

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They wouldn't have all entered the genome at the same time.

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Have you ever seen a child whose mother caught a viral disease early in her pregnancy? It destroys the stem cell.

Dredge:

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Junk No Longer: ERVs Are “Integral” and “Important Components” of Immune Responses

Taq:

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The usual red herring.

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What's the matter, it doesn't fit your narrative.

Kleinman:

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Have you ever seen a child whose mother caught a viral disease early in her pregnancy? It destroys the stem cell.

Taq:

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We can observe ERV's in the koala genome that is inherited as an ERV, and it is the same sequence as that found in the circulating retrovirus that is currently infecting koalas. We can see it in action. If what you say is true, then these observations would not be possible, and yet there they are.

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You have never seen a child whose mother caught a viral disease early in her pregnancy. You have no idea what happens to that child.

Kleinman:

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You have never seen a child whose mother caught a viral disease early in her pregnancy. You have no idea what happens to that child.

Taq:

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Does every single child in this situation die? No.
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You have never offered a scintilla of evidence demonstrating that the insertion of viral sequence into a germline cell results in the loss of the fetus in every single case.

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The birth defects are horrendous and that is from a single viral infection. Now you want to claim there are hundreds of thousands of viral infections and they are all inactivated. This is stupid to no end. These may superficially look like viral sequences but they are something else.

Kleinman:

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The birth defects are horrendous and that is from a single viral infection. Now you want to claim there are hundreds of thousands of viral infections and they are all inactivated. This is stupid to no end. These may superficially look like viral sequences but they are something else.

Taq:

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You have never offered a scintilla of evidence demonstrating that the insertion of viral sequence into a germline cell results in the loss of the fetus in every single case.

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What's the probability of a germ line cell being infected hundreds of thousands of times and there not being damage to that cell? This is the kind of nonsense you have fallen for.

Dredge:

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The following article is a bit old (2011), but I thought you might find it interesting nevertheless:
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Do Shared ERVs Support Common Ancestry? – Evolution News

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That's interesting, but why should Taq read it, it doesn't fit his narrative.