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Author | Topic: Any practical use for Universal Common Ancestor? | |||||||||||||||||||||||||||||||||||||||
PaulK Member Posts: 17914 Joined: Member Rating: 6.9 |
Caffeine has provided a better answer than I can but here’s a bit more to think about.
quote: Well let’s start by pointing out that your ideas of which traits are “superficial” seems to be based purely on convenience to your position. Sharper claws is not a superficial trait to you, but all the variations in trilobite eye structure are. On any objective assessment that is utterly ridiculous. Next, while parents and children will generally be similar they will not be identical. There will be small differences in the genome that are not inherited from the parents. Some of these differences will have a phenotypic effect and some of these will persist and eventually take over a population of the descendants - many generations down the line. So, it is not that there is no change - there is slow, cumulative change within a population.
quote: This is not a sensible question. How can you ask about specifics when dealing with the general situation ? There can be biochemical changes, changes in colouration or shape, hair might be gained or lost. The hooded crow and carrion crow seem to be separating based on nothing more than sexual selection - the plumage differences being enough to drive them apart. No doubt they will slowly diverge, since there is very little, if any, gene flow between the populations. But how they will diverge is simply not predictable. We can’t know which mutations will occur or which of those will be advantageous over the next few millennia (and I expect it will take longer than that for them to become greatly distinct)
quote: As caffeine points out, that isn’t true. It would take adjustments to regulatory sequences to affect the development of the paw.
quote: A genome is not a blueprint. It does not describe the morphology at all. It is more like instructions to grow the creature. Make this protein under these conditions is the basic level. And adjusting those instructions produces the sort of differences we see.
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PaulK Member Posts: 17914 Joined: Member Rating: 6.9
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quote: So what is your reason for using an obvious double standard ? I think the fact that your arguments would collapse without it shows adequate reason for my conclusion. Note that this is a highly relevant point since you claim that the minor differences between a human hand and a chimpanzee’s require new genes.
quote: Did you admit that there are differences in the genome that are not inherited from the parents ? Did you admit that these differences can include phenotypic changes ? Did you admit that these new traits can spread to entire populations ? If not then you are the one failing to pay attention.
quote: A new trait coming from a mutation would seem to fit.
quote: Since the points you are objecting to are obviously relevant it seems that the reality is that you don’t want an answer.
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PaulK Member Posts: 17914 Joined: Member Rating: 6.9 |
quote: Blueprint is not a good metaphor, because the genes do not directly represent the phenome in any way - map isn’t exactly better for the same reason. Recipe or instruction booklet are not as bad, but still potentially misleading. The relationship between gene and phenotype depends very, very much on the trait you are talking about. The structure of a protein depends directly on the genes. But the further you get away from that the less and less sense it makes to talk about individual genes instead of the whole system - including the environment where development is taking place, especially if it is an egg or a womb. Look up “maternal effect genes”.
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PaulK Member Posts: 17914 Joined: Member Rating: 6.9 |
quote: No, it’s the truth. Really, this aggressive lying is not helping anyone, least of all you.
quote: But I am not picking “one gene” at all, and it certainly isn’t true that only one gene doesn’t fit with the idea that a gene directly causes a trait, so your accusation of fraud is another lie. Researchers may identify genes by effects - but that may mean no more than that one particular allele is associated with a particular trait in a single species. Until you understand the relationship between the allele and the trait you can’t say any more.
quote: Of course, since you believe in the Creationist “kind”concept you don’t really believe that it is impossible at all. Nor have you identified any real issues that would prevent genetic changes accumulating through drift and selection until a population of descendants has become a distinct species. So the general question is answered - and your claim of impossibility remains unsupported.
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PaulK Member Posts: 17914 Joined: Member Rating: 6.9 |
quote: You’ve identified no real barriers at all.
quote: You ask a general question you get general answers. Which you can’t refute. And the fact that you obviously don’t want answers and object strongly to any explanation of how genes relate to traits is hardly helpful.
quote: We don’t have to assume that dishonesty on your part is obstructing reasonable discussion - we know it for a fact.
quote: People have. You don’t like it at all.
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PaulK Member Posts: 17914 Joined: Member Rating: 6.9 |
Some genetic differences between humans and chimpanzees, found here
For example, one gene called the MYH16 contributed to the growth and development of very huge jaw muscles in apes. In people, MYH16 has actually been deactivated. Most people have lost another muscle-related gene called ACTN3. Humans with two working versions of the gene are basically overrepresented among the elite sprinters while the nonworking versions are overrepresented among the endurance runners. On the other hand, chimps and other nonhuman primitives have only a working version.
Wikipedia explains MYH16
The MYH16 gene encodes a protein called myosin heavy chain 16, which is a muscle protein in mammals. At least in primates, it is a specialized muscle protein found only in the temporalis and masseter muscles of the jaw.[1][2] Myosin heavy chain proteins are important in muscle contraction, and if they are missing, the muscles will be smaller.[1] In non-human primates, MYH16 is functional and the animals have powerful jaw muscles. In humans, the MYH16 gene has a mutation that causes the protein not to function.[3]
And ACTN3
Alpha-actinin is an actin-binding protein with multiple roles in different cell types. This gene expression is limited to skeletal muscle.
However it must be remembered that chimpanzees are very similar to humans in many ways E.g.
The internal anatomy is also almost the same between chimps and humans. The circulatory, respiratory, and digestive systems of chimps and humans look almost the same and work in nearly identical ways.
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PaulK Member Posts: 17914 Joined: Member Rating: 6.9
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quote: The pathways already invoked for microevolution seem to be adequate. Especially as they include gaining and losing genes. Added: and for an unusual but relevant case, see this thread and the article linked in the OP. Message 1 If you have any concrete objections it is rather surprising that you haven’t seriously raised them.
quote: There is nothing special about a “species-specific genome” that means that it can’t change. Indeed discussion around that point seems meaningless. The “species-specific genome” is based on the individual genomes of the members of that species and will change as the membership of that species changes. If a sub-population is isolated it will diverge from the rest of the species - drift alone guarantees that. The changes that occur must be counted when identifying the “species-specific genome” right up until that population is considered to be a different species when it isn’t. Or to put it simply there is absolutely nothing special about “species specific genomes” that suggest any problems in going from one to another. Changes occur, some of them will end up in new species while not being considered as belonging to the parent species (even if they were present for a time) but there is nothing special about those changes. They were just those that happened to occur as part of a speciation event rather than just adding to the diversity of the main population. Edited by PaulK, : No reason given.
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PaulK Member Posts: 17914 Joined: Member Rating: 6.9 |
quote: Simple assertions aren’t really adequate answers.
quote: In practical terms that is true, but that’s not because there is anything special about the genome - and it doesn’t mean that that genome is constant. Variations can and do come and go. No meaningful objection there.
quote: Presumably you mean traits that don’t belong to the species - since shared traits are ubiquitous. But this is also meaningless as an objection. Any heritable trait found in the species “belongs” to the species, and the genome will be involved in it’s existence.
quote: Then that is already answered. If new traits appear through mutation - as we know can happen - then they will “belong” to the species and be the product of the genome. Even though the genome lacked the capability to produce the trait before.
quote: Perhaps you can explain the differences between a chimp fingernail and a human fingernail and explain the role of the genome in producing those differences.
quote: But you claim to be able to show that it is impossible. Asking for details that I’m not in a position to know doesn’t show that. It’s just a rhetorical trick. An attempt to cover up the fact that you cannot show any real barrier. I can be sure that if I asked you to give a similarly detailed explanation of how a single species genome could produce the much bigger variations seen in trilobites - in the absence of massive artificial selection - you would complain loudly. But it is no less fair than your demand.
quote: You mean that you want us to repeat the examples of the pocket mice and the peppered moth and the Scottish fold cat all over again ?
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PaulK Member Posts: 17914 Joined: Member Rating: 6.9
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quote: But I don’t know that there is any significant difference between a chimp fingernail and a human fingernail at all - let alone one that is out of the reach of mutation. And neither do you. And that makes it pointless as an example.
quote: And - unless we get into quibbles - we also know that it is a meaningless tautology that doesn’t help your argument at all. So let’s stop wasting time with rhetorical games. If you have a real case, then make it. If you don’t then cut out the aggression and honestly admit it.
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PaulK Member Posts: 17914 Joined: Member Rating: 6.9
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quote: You claim that there are real obstacles and therefore it is on you to show that there are. But you don’t. What is wrong with the examples you have been given? In the absence of real objections they do meet your demand here. Indeed, in the absence of any real obstacles you lose there, too. Edited by PaulK, : No reason given.
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PaulK Member Posts: 17914 Joined: Member Rating: 6.9
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quote: You do realise that this is a pointless question which can’t possibly help you ? The reason has been given over and over again. Genomes change over time. They are moving targets. Nobody is suggesting that a (current) human genome would produce a chimp or vice versa. You have to show that the changes are impossible. Arguing that the genome for a species produces that species is a complete irrelevance.
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PaulK Member Posts: 17914 Joined: Member Rating: 6.9 |
quote: You’d always get something that belonged to the mutated genome, but that doesn’t exactly help you, does it ?
quote: And this is pointless. We are talking about changes to the genome. Just going back to an unchanged genome all the time evades the issue. We have proof that the genome does change. We know that the differences between chimp and human are compatible with the mechanisms of change. We have other evidence that chimp and human both evolved from a common ancestor (largely on the human side since chimps live in an environment that is very destructive to their remains). You have got a bald declaration that it can’t happen and instead of talking about it you go back to the current genomes every time. We are way ahead of you. Demanding more of us when you can’t even match what we have is less than fair.
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PaulK Member Posts: 17914 Joined: Member Rating: 6.9
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quote: Aside from the fact that mutations are pretty ordinary that is basically what we’ve been saying.
quote: That may be what you mean by microevolution but scientists - and I expect most creationists count mutations as microevolution too. You probably should, too because you don’t want to define them as macroevolution - which would be the only reasonable alternative.
quote: Now you are not making sense. Mutations are changes to the genome. They include disabling and deleting genes, and even the formation of duplicate genes - or more rarely the creation of new genes.
quote: Not really. Most weird anomalies are developmental problems which aren’t even caused by mutations. Many mutations have relatively little effect - as you would expect for small chemical variations in proteins. To understand more you would need to know more about developmental biology than you do.
quote: But this comes down to what should be considered an error. If we ignore all mutations without phenotypic effect - and there are huge numbers of those - any beneficial or neutral mutation should be considered a success, not an error. Your point is just an uninformed guess.
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PaulK Member Posts: 17914 Joined: Member Rating: 6.9 |
quote: Well obviously changes to the genome are in the genome. Where else would they be ?
quote: You are going to have to explain that because it is very unclear what it includes or excludes. Changes to the genome can obviously change what it does. We’ve seen examples. But any such changes are necessarily within the new genome and ARE what it does. So how different does the new genome have to be from the old genome to qualify ? Do very small changes count ? If they don’t what happens when a number of small changes add up to a larger change ? Does that count ?
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PaulK Member Posts: 17914 Joined: Member Rating: 6.9 |
quote: If you mean something that the old genome couldn’t do then the pocket mice and the Scottish Fold cat qualify. But you obviously can’t mean something that the new genome can’t do. So I just have to repeat my question. What changes count? Obviously you must be dismissing some changes, but why ?
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