Review and Confirm The Mathematical Proof of God

The cross has either 2 parts or 4 parts, depending on how you view it. The cross is an ancient Roman method of torture and execution. It lacks the features required to be a planet, such as clearing its orbit of other bodies and moving in the planetary disk of the solar system. Any such "proof" could be made between math and any given fairy tale.

The hands and feet are not joined as one. Those aren't the key characteristics of planets because there are many, many other objects that have those features, such as many meteors. Only in your imagination.

The Judeo/Christian God isn't fixed into the clock either. The hands are at 3 and 9 which added together makes 12. 12 divided 3 or 9 is 4 or 1 and 1/3. Neither of those are the trinity. It fails.

Right. And if you add them together you get 12. When you divide by 3 you get 4, and this is not the trinity. Therefore, God is not fixed into the clock.

I already showed you the mathematical proof that it doesn't align. 3+9 = 12. 12/3=4. That's a failure to align. 4 is not 3 which is the trinity.

3*1 = 3. That's not what the clock shows. The clock shows 4 because 3+9 is 12, and when you divide by 3 you get 4. This disproves your entire proof.

It doesn't fit into the Time Clock. I already disproved that.

I have one for you.Do you know what pareidolia is? Yes/No

"Pareidolia (/ˌpærɪˈdoʊliə, ˌpɛər-/;[1] also US: /ˌpɛəraɪ-/)[2] is the tendency for perception to impose a meaningful interpretation on a nebulous stimulus, usually visual, so that one detects an object, pattern, or meaning where there is none. Pareidolia is a type of apophenia."
Pareidolia - Wikipedia

Dunning-Kruger indeed. He is not only stuck on the ignorance peak, but incapable of learning anything new. He still thinks new proteins evolve by random assembly of amino acids, for crying out loud. Don't they teach RNA transcription and protein translation in high school biology? Perhaps they don't.The lesson for other readers: learn the basics of cell biology before attempting to lecture biologists.

So says the theist who pretends to lecture biologists on proteins without knowing the most basic concepts of cell biology. I sometimes agree with crazy people. I find it makes them go away.For example, you claim titin has to have a precise sequence. There are over 15,000 sequence variants of titin just in humans. How do you explain this? Yet another claim you will never be able to defend once we dig into the actual facts. I would assume this is based on the Axe and Gauger paper where they start with a single peptide sequence and then look to see how many mutations it would take to get beta-lactamase activity, a single specific function. Using these same criteria, there are only around 5 proteins in the entire human body that have function. Titin doesn't have beta-lactamase activity, so is it a non-functional protein?What you don't seem to realize is that the same function can arise from many different genetic backgrounds, and there are many, many possible functions. If what you claim is true then we should have all died from bacterial and viral infections because none of the antibodies we produce would function. Out of the billions of unique, randomly assembled binding domains in our collection of antibodies there are millions that have function which is why we are able to fight off infections. Of course, B-cell maturation, V(D)J recombination, and antibody selection are probably processes you don't understand since you can't even grasp where proteins come from.

I specifically stated evolution, because titin evolved. Titin didn't appear until there were multicellular organisms with muscles. This is about 3 billion years after life first appeared on Earth.The first titin protein would have been transcribed from DNA and translated from RNA. It wouldn't have been randomly assembled by connecting amino acids.Added in edit:Wiki is your friend:

quote:

---

The titin domains have evolved from a common ancestor through many gene duplication events.[34] Domain duplication was facilitated by the fact that most domains are encoded by single exons. Other giant sarcomeric proteins made out of Fn3/Ig repeats include obscurin and myomesin. Throughout evolution, titin mechanical strength appears to decrease through the loss of disulfide bonds as the organism becomes heavier.[35]

​

Titin A-band has homologs in invertebrates, such as twitchin (unc-22) and projectin, which also contain Ig and FNIII repeats and a protein kinase domain.[30] The gene duplication events took place independently but were from the same ancestral Ig and FNIII domains. It is said that the protein titin was the first to diverge out of the family.[28] Drosophila projectin, officially known as bent (bt), is associated with lethality by failing to escape the egg in some mutations as well as dominant changes in wing angles.[36][37][38]

​

Drosophila Titin, also known as Kettin or sallimus (sls), is kinase-free. It has roles in the elasticity of both muscle and chromosomes. It is homologous to vertebrate titin I-band and contains Ig PEVK domains, the many repeats being a hot target for splicing.[39] There also exists a titin homologue, ttn-1, in C. elegans.[40] It has a kinase domain, some Ig/Fn3 repeats, and PEVT repeats that are similarly elastic.[41]
Titin - Wikipedia

---

So there are even vertebrate titin homologs in invertebrates. What in the world are you talking about??????? We have nearly the same gene content as other apes and primates. Titin is found in many vertebrate species other than humans, so it was found in the common ancestor of vertebrates.